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It is suitable for chronic reversible airway obstructive diseases such as bronchial asthma, but is not suitable for rapid relief of bronchospasm. Therefore, budesonide should not be used alone to treat status asthmaticus or other acute asthma exacerbations, which require intensive treatment. Special caution is required in patients who are being switched from oral steroids to budesonide therapy. During asthma exacerbations or severe attacks, patients require additional oral steroids. It is recommended that these patients carry a warning card (see Clinical Management: Patients Dependent on Oral Corticosteroids). Patients who have previously received high-dose systemic steroid therapy may relapse into early allergic symptoms, such as rhinitis and eczema, when switching from oral therapy to budesonide because of the lower systemic steroid effect of budesonide. During long-term high-dose treatment with budesonide, hematological and adrenal function monitoring is recommended.

Usage and dosage of budesonide: 1. Take budesonide about half an hour before meals, and swallow it whole with a large amount of liquid (such as a glass of water).

2. Treatment time for active Crohn's disease and collagenous colitis should be limited to 8 weeks.

3. Treatment to maintain remission of autoimmune hepatitis should be continued for at least 24 months. Its termination is possible only if biochemical remission is sustained and signs of inflammation are absent in liver biopsy.

4. Termination of treatment: It should not be stopped suddenly, but gradually canceled (tapered). During the first week, the daily dose should be reduced to two capsules, one in the evening and one at night. During the second week, take only one capsule in the morning. Treatment can then be stopped.

It needs to be used under the advice of a doctor. For specific medication information of budesonide, patients are advised to consult their attending doctor. It is generally believed that there will be no systemic adverse reactions of glucocorticoids. Ketoconazole can increase the plasma concentration of budesonide. The mechanism of action of budesonide may be to inhibit the metabolism of budesonide mediated by cytochrome P4503A.